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Publications

An adverse tumor-protective effect of IDO1 inhibition.

Kenski JCN. et al, (2023), Cell Rep Med, 4

AMPK phosphorylates ZDHHC13 to increase MC1R activity and suppress melanomagenesis.

Sun Y. et al, (2023), Cancer Res

Sublethal cytochrome c release generates drug-tolerant persister cells.

Kalkavan H. et al, (2022), Cell, 185, 3356 - 3374.e22

The MITF regulatory network in melanoma.

Chauhan JS. et al, (2022), Pigment cell & melanoma research, 35, 517 - 533

Reciprocal Regulation of BRN2 and NOTCH1/2 Signaling Synergistically Drives Melanoma Cell Migration and Invasion.

Fane ME. et al, (2022), The Journal of investigative dermatology, 142, 1845 - 1857

MC1R Functions, Expression, and Implications for Targeted Therapy.

Guida S. et al, (2022), The Journal of investigative dermatology, 142, 293 - 302.e1

Connecting Metabolic Rewiring With Phenotype Switching in Melanoma.

Falletta P. et al, (2022), Frontiers in cell and developmental biology, 10

TBX2 controls a proproliferative gene expression program in melanoma.

Lu S. et al, (2021), Genes & development, 35, 1657 - 1677

TOP1 modulation during melanoma progression and in adaptative resistance to BRAF and MEK inhibitors

Oliveira ÉAD. et al, (2021), Pharmacological Research, 173, 105911 - 105911

TBX3 Promotes Melanoma Migration by Transcriptional Activation of ID1, which Prevents Activation of E-Cadherin by MITF.

Peres J. et al, (2021), The Journal of investigative dermatology, 141, 2250 - 2260.e2

BRN2 is a non-canonical melanoma tumor-suppressor.

Hamm M. et al, (2021), Nature communications, 12

Establishment of a fluorescent reporter of RNA-polymerase II activity to identify dormant cells.

Freter R. et al, (2021), Nature communications, 12

The protective role of MC1R in chromosome stability and centromeric integrity in melanocytes.

Li X. et al, (2021), Cell death discovery, 7

MITF induces escape from innate immunity in melanoma.

Sánchez-Del-Campo L. et al, (2021), Journal of experimental & clinical cancer research : CR, 40

Organotypic Models in Drug Development "Tumor Models and Cancer Systems Biology for the Investigation of Anticancer Drugs and Resistance Development".

de Oliveira ÉA. et al, (2021), 265, 269 - 301

Acriflavine, a Potent Inhibitor of HIF-1α, Disturbs Glucose Metabolism and Suppresses ATF4-Protective Pathways in Melanoma under Non-Hypoxic Conditions

Martí-Díaz R. et al, (2020), Cancers, 13, 102 - 102

The transcription factor NRF2 enhances melanoma malignancy by blocking differentiation and inducing COX2 expression.

Jessen C. et al, (2020), Oncogene, 39, 6841 - 6855

Tuning Transcription Factor Availability through Acetylation-Mediated Genomic Redistribution

Louphrasitthiphol P. et al, (2020), Molecular Cell, 79, 472 - 487.e10

Paradoxical activation of AMPK by glucose drives selective EP300 activity in colorectal cancer.

Gutiérrez-Salmerón M. et al, (2020), PLoS biology, 18

ABCB5is activated by MITF and β‐catenin and is associated with melanoma differentiation

Louphrasitthiphol P. et al, (2020), Pigment Cell & Melanoma Research, 33, 112 - 118

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