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Two distinct subsets of γδ T cells that produce interleukin 17 (IL-17) (CD27(-) γδ T cells) or interferon-γ (IFN-γ) (CD27(+) γδ T cells) develop in the mouse thymus, but the molecular determinants of their functional potential in the periphery remain unknown. Here we conducted a genome-wide characterization of the methylation patterns of histone H3, along with analysis of mRNA encoding transcription factors, to identify the regulatory networks of peripheral IFN-γ-producing or IL-17-producing γδ T cell subsets in vivo. We found that CD27(+) γδ T cells were committed to the expression of Ifng but not Il17, whereas CD27(-) γδ T cells displayed permissive chromatin configurations at loci encoding both cytokines and their regulatory transcription factors and differentiated into cells that produced both IL-17 and IFN-γ in a tumor microenvironment.

Original publication

DOI

10.1038/ni.2702

Type

Journal article

Journal

Nature immunology

Publication Date

10/2013

Volume

14

Pages

1093 - 1100

Addresses

Instituto de Medicina Molecular, Faculdade de Medicina, Universidade de Lisboa, Lisbon, Portugal.

Keywords

T-Lymphocyte Subsets, Th1 Cells, Animals, Mice, Knockout, Mice, Receptors, Antigen, T-Cell, gamma-delta, Histones, Cytokines, Gene Expression Profiling, Cell Differentiation, Gene Expression Regulation, Epigenesis, Genetic, Methylation, Female, Genome-Wide Association Study, Th17 Cells, Transcriptome